This book is a collection of Mark King's best known instrumental tracks as recorded with Level 42. The book also contains transcriptions of three of Mark's live bass solos and a detailed style analysis. ISBN: 978-0-9557981-7-7 Number of Pages: 157
These transcriptions have all been done for gigs/personal research or requests from private students. Most are note-for-note transcriptions of bass parts, some are chord charts and others are a combination of the two.
Level 42 Bass Transcriptions Pdfl
Awesome site Tom. As a fellow bassist, you have motivated me to post my transcriptions. I assume since you (and I) are/would be giving them away that you are not worried about the copyright people. My charts are all for contemporary praise music. Ron Wright
Ron Carter, the icon of walking jazz bass lines, has now re-invented the way transcriptions are written. The method is called "Chartography" (often misspelled as Cartography)and musicians will be in awe of how much they can learn when studying this completely new innovation.
The fold change in the transcription levels of the four redox genes txn1, gpx1, txnrd3, and txnr2 in Phase A is shown in Fig. 2a, b, c, and d, respectively. As was the case for the reductase activities, the correlation between the transcription of the genes and the Hg in the feed was negative considering the four groups together (Table 3). However, the median values for the transcription of the hepatic selenogenes in A2, fed as A1 an excess of Se over Hg, were higher than that of A1 for txn1 and gpx1, and it was only slightly different for txnrd2 and txnrd3 (Fig. 2). Under an excess of Hg, on the other hand, the transcriptions of all the genes were downregulated (Fig. 2).
The transcription levels of txn1, gpx1, txnrd3, and txnrd2 in Phase B are shown in Fig. 2 and in Table 5 and follow the same pattern as in Phase A for fish fed diets with a molar excess of Hg over Se: exposure to high Hg ratios downregulated txn1, txnrd3, and txnrd2 and upregulated gpx1. With respect to B1, the downregulation in the transcriptions of B2 was in general less severe than in B3 (Table 5), indicating a protective effect of the inclusion of TH in the diet of these fish. The upregulation of gpx1 in both groups may indicate complementarity among selenogenes from the antioxidant pathways. The glutathione system, particularly glutathione reductase, has been reported to complement reduced Trx and TrxR activities in the liver of zebra seabreams during Hg exposure [35]. Although that study did not examine the mRNA levels, our results support that complementarity at the transcriptional level by the negative correlation between the (lowered) activity of both reductases (Fig. 1c, d) and the (upregulated) transcription of gpx1 (Fig. 2g).
Furthermore, although no studies have been published regarding sea bass, pre- and peri-natal exposure to Hg has effects on selenoprotein activity that may be age-, sex-, and species-dependent. Thus, glutathione peroxidase activity in rats was more than double in hepatic mitochondria from females than from males of the same age [46], and in four species of freshwater fish (Ambloplites rupestris, Lepomis auritus, Lepomis gibbosus, and Lepomis macrochirus), the rate of Hg accumulation was found to be significantly faster and the mean Hg levels to be higher in females than in males after the onset of reproduction, although not earlier. On the other hand, previous works had found that males contained higher Hg levels than females in other species [47] and references therein). In mice, exposure to MeHg+ during prenatal and lactational periods influenced in a sex- and brain anatomical structure (cerebrum and cerebellum)-specific manner the responses of TrxR, Trx, and GPx [39].
In the present work, the ubiquitousness of Hg (found both in the feed and in the wild hake) not only made it impossible to provide a real control group fed no Hg at all, but also suggests that the broodstock from which these sea bass originated had also been fed Hg-containing feeds. Accordingly, the experimental sea bass used in this work had most likely already been exposed to Hg contamination in ovo, albeit probably to low levels and in conditions of a high Se/Hg molar ratio (given the Se/Hg molar ratios of the feed). The lack of gonad development in our fish did not allow us to determine their sex. Consequently, although both pre-hatch exposure to MeHg+ and sex may have influenced the variability of our results, we cannot determine whether they actually did it or to what extent. Further studies should be carried out in the future to clarify these issues. 2ff7e9595c
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